Você está em:Start > Publications > View > Titin phosphorylation deficit and high cardiomyocyte stiffness in an animal model of metabolic risk induced heart failure with preserved ejection fraction.
Titin phosphorylation deficit and high cardiomyocyte stiffness in an animal model of metabolic risk induced heart failure with preserved ejection fraction.
Titin phosphorylation deficit and high cardiomyocyte stiffness in an animal model of metabolic risk induced heart failure with preserved ejection fraction.
Type
Other Publications
Year
2013
Authors
Hamdani, N
(Author)
Other
The person does not belong to the institution.
The person does not belong to the institution.
The person does not belong to the institution.
Without AUTHENTICUS
Without ORCID
The person does not belong to the institution.
The person does not belong to the institution.
The person does not belong to the institution.
Without AUTHENTICUS
Without ORCID
The person does not belong to the institution.
The person does not belong to the institution.
The person does not belong to the institution.
Without AUTHENTICUS
Without ORCID
Linke, WA
(Author)
Other
The person does not belong to the institution.
The person does not belong to the institution.
The person does not belong to the institution.
Without AUTHENTICUS
Without ORCID